Jamieson described the development of MF from the context with the JAK/STAT community. “This pathway is upregulated in the majority of patients Using these myeloproliferative neoplasms. In a few, It is based upon a mutation in JAK2 that prevents the gene from staying turned off; in Some others, a mutation https://martinwishr.blogofoto.com/57196400/mouse-an-overview