Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. Alternatively, our ability to rescue SERT perform in KI midbrain synaptoneurosomes from the inhibition of FAK suggests elevated FAK signaling downstream with the Pro32Pro33 mutant, as verified by improved pFAK localization in 5-HT synapses. Our information https://chaplino642qdm4.loginblogin.com/profile